Sub-section Headers

Once again we see the contrast between informative versus indicative, that is what was done versus what did we learn?

Compare these two versions

Correlating microbial community profiles with geochemical data in highly stratified sediments from the Arctic Mid-Ocean Ridge The HIF Signaling Pathway in Osteoblasts Directly Modulates Erythropoiesis through the Production of EPO
Core Descriptions. Generation of Mice with HIF-1 and HIF-2 Overstabilization and Inactivation in Osteoprogenitor Cells
Composition and Diversity of the Microbial Community. Augmented HIF Activity in Osteoprogenitors Expands the HSC Niche in the Bone Marrow
Determination of Bacterial and Archaeal Abundance. Selective Expansion of the Erythroid Lineage in OSX-VHL Mutant Mice Leads to the Development of HIF-Dependent Polycythemia
Principal Components and Correlation Analyses. Increased Hematocrit in OSX-VHL Mice Is EPO Dependent and Associated with Increased EPO Expression in Bone and Decreased EPO Expression in the Kidney
Distribution of the Most Abundant Bacterial Phyla. Loss of HIF-2 in Osteoblasts Impairs EPO Expression and Numbers of Erythroid Progenitors in the Bone Marrow but Does Not Cause Anemia
Distribution of the Most Abundant Archaeal Phyla. Modulation of the PHD/VHL/HIF Pathway in Osteoblasts Is Sufficient to Induce EPO and Protect from Anemia
Table 1: A comparison of informative and indicative sub-section headings in the results section

In Table tbl-subheader-results we can …

Correlating microbial community profiles with geochemical data in highly stratified sediments from the Arctic Mid-Ocean Ridge
Core Descriptions.
Composition and Diversity of the Microbial Community.
Determination of Bacterial and Archaeal Abundance.
Principal Components and Correlation Analyses.
Distribution of the Most Abundant Bacterial Phyla.
Distribution of the Most Abundant Archaeal Phyla.
The HIF Signaling Pathway in Osteoblasts Directly Modulates Erythropoiesis through the Production of EPO
Generation of Mice with HIF-1 and HIF-2 Overstabilization and Inactivation in Osteoprogenitor Cells
Augmented HIF Activity in Osteoprogenitors Expands the HSC Niche in the Bone Marrow
Selective Expansion of the Erythroid Lineage in OSX-VHL Mutant Mice Leads to the Development of HIF-Dependent Polycythemia
Increased Hematocrit in OSX-VHL Mice Is EPO Dependent and Associated with Increased EPO Expression in Bone and Decreased EPO Expression in the Kidney
Loss of HIF-2 in Osteoblasts Impairs EPO Expression and Numbers of Erythroid Progenitors in the Bone Marrow but Does Not Cause Anemia
Modulation of the PHD/VHL/HIF Pathway in Osteoblasts Is Sufficient to Induce EPO and Protect from Anemia